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. 2021 Sep 25;2021:8457521. doi: 10.1155/2021/8457521

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Copyright © 2021 Rui Xiong et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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The possible mechanism of targeting ferroptosis enhancing antilung cancer. (IFNγ: interferon-γ; GSH: glutathione; LncRNA: long noncoding RNA; IR: irradiation; STYK1/NOK: serine threonine tyrosine kinase 1/novel oncogene with kinase domain; GPX4: glutathione peroxidase 4; Nrf2: nuclear factor erythroid 2-related factor 2; EGLN1/c-Myc: Egl Nine Homolog 1/c-Myc; LSH: lymphoid-specific helicase; GINS4: Go, Ichi, Nii, and San complex subunit 4; ACSL4: acyl-CoA synthetase long chain family member 4; FSP1: ferroptosis suppressor protein 1).