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. 2021 Oct 3;27:124. doi: 10.1186/s10020-021-00367-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — MiR-146b-5p-mediated sh-MINCR protected against lipopolysaccharide (LPS)-induced acute lung injury (ALI). A total and neutrophil counts in bronchoalveolar lavage fluid (BALF) mice. B mouse BALF myeloperoxidase (MPO) activity. C hematoxylin and eosin staining. Scale bar = 50 μm. D terminal deoxynucleotidyl transferase biotin-dUTP nick end labeling staining was used to detect apoptosis. Scale bar = 50 μm. E F4/80 IHC staining. Scale bar = 50 μm. F quantification of levels of TNF (tumor necrosis factor)-α, IL (interleukin)-6, and IL-10 were examined by enzyme-linked immunosorbent assay. *p < 0.05, **p < 0.01, ***p < 0.001 vs. Ctrl group; ^#p < 0.05 vs. LPS. N = 10.