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. 2021 Sep 20;12:722730. doi: 10.3389/fphar.2021.722730

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Copyright © 2021 Chun, Lee, Ahn, Baek, Yi, Shin and Jung.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SHCGT regulated gut microbiome-related metabolites and short-chain fatty acids and reduced endotoxin levels. (A) Concentrations of short-chain fatty acids, including butyrate, isobutyrate, propionate, and valerate. SHCGT significantly increased the isobutyrate, propionate, and valerate levels in mice with DNCB-induced AD. (B) Change in plasma endotoxin levels. The endotoxin level was lower in the group with SHCGT-treated AD than in the sham and AD groups. *p < 0.05, **p < 0.01 vs. sham; ^# p < 0.05, ^## p < 0.01 vs. AD. AD, atopic dermatitis; DNCB, 2,4-dinitrochlorobenzene; SHCGT, Sihocheonggan-Tang.