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. 2021 Oct 5;12:5816. doi: 10.1038/s41467-021-26088-z

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — The CK receptors AHK2 and AHK3 as well as the type-B response regulators ARR1, ARR10, and ARR12 are activated mainly by tZ. Activation of CK signaling in the shoot results in an increase of miR172 abundance. MiRNA activation may be achieved directly by type-B ARRs or involve co-action of SPLs. TOE1 and TOE2 are repressed posttranscriptionally upon miR172 increment, enabling the transition from the juvenile to the adult vegetative phase.