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. 2021 Oct 6;12:5848. doi: 10.1038/s41467-021-26153-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Tissues assayed by this study. b, c Average peak number and genome coverage for each epigenetic mark in each tissue. d The Pearson correlations among assays, tissues, and biological replicates (P348 and P350) based on the normalized signal in 1 kb windows stepped across the whole genome. e Average epigenetic mark signal proximal to protein-coding genes. TSS transcription start site, TES transcription end site. f Epigenetic signal at the MYO1A locus according to different assays and in different tissues. Vertical scale of UCSC tracks shows normalized signal from 0 to 200 for RNA-seq, 0 to 100 for H3K27ac and H3K4me3, and 0 to 50 for other marks and ATAC-seq.