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. 2021 Oct 9;12:107. doi: 10.1186/s40104-021-00628-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Correlation between the ileal mycobiota composition and NE severity. Chickens were sequentially infected with E. maxima and C. perfringens to induce NE. Intestinal lesions in individual chickens were scored. Spearman correlation analysis was performed between the ileal mycobiota profile and the intestinal lesion score and weight loss. A Spearman correlation at family, genus, and amplicon sequence variant (ASV) levels. B Heatmap displaying log2 transformations of fold changes in fungal abundance in NE chickens at the family, genus, and ASV levels, relative to mock-infected controls. Significance of Spearman correlations was subjected to FDR correction using the Benjamini-Hochberg procedure. *FDR < 0.05, **FDR < 0.01