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. 2021 Sep;9(18):1476. doi: 10.21037/atm-21-4652

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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TE-MenSCs regulates the phenotype of EECs by activating the EGF/Ras p21 pathway. (A) Effect of EGF treatment on cell invasion of EECs. (B) Effect of EGF treatment on cell proliferation of EECs (×400). (C) Effect of EGF treatment on the mRNA expression of MAPK p38 and Ras p21 in EECs as determined by RT-PCR. (D) Effect of EGF treatment on the protein expression of MAPK p38 and Ras p21 in EECs as determined by western blotting. TE-MenSCs, menstrual stem cells from thin endometrium; NTE-MenSCs, menstrual stem cells from non-thin endometrium; EECs, endometrial epithelial cells; mRNA, messenger RNA; RT-PCR, reverse transcription polymerase chain reaction.