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. 2021 Oct 14;11:20476. doi: 10.1038/s41598-021-00064-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Twenty-six-year-old male with no prior co-morbidities; admitted with severe RT-PCR proved SARS-CoV-2 pneumonia, (A) as shown on the chest radiogram. This patient, who was on mechanical ventilation, went into cardiac arrest. Brain MRI was performed 4 days post arrest due to low GCS (= 6), revealed global hypoxic-ischemic encephalopathy. (B) Axial FLAIR image, at the level of the BG, showed decreased signal intensity in the thalami, while (C) corresponding DWI image showed lack of hyperintensity in both thalami, in addition to the caudate and putamen nuclei, findings that represent pseudo normalization. (D) Also, corresponding ADC map showed hyperintensity in the locations cited in (B). (E) Reconstructed maximum intensity projection magnetic resonance angiography (MRA) image shows no large vessel occlusion.