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. 2021 Oct 15;19:322. doi: 10.1186/s12951-021-01062-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 12 — In vivo antitumor activity by G-PTX. A Fabrication of ginsenoside-anchored liposome and surface glycosyl of ginposome for active-targeting ability to GLUT1 receptor on the surface of tumors. B The suppressed tumor growth of patient-derived xenograft tumor models treated with G-PTX. C The verification of G-PTX active targeting, G-PTX uptake was reduced via inhibiting GLUT1 by siRNA transfection. D The expression of autophagy-related proteins of HGC-27 cells indicating the reversal of drug resistance. Reprinted with the permission from Ref [27].

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