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. 2021 Oct 14;14:136. doi: 10.1186/s13048-021-00888-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Circ_0061140 silencing enhanced the inhibitory effects of PTX on tumor growth in vivo. (A and B) The effects of circ_0061140 absence on the volume and weight of tumors after PTX treatment were revealed. (C and D) QRT-PCR was employed to determine the impacts of circ_0061140 silencing on the expressions of circ_0061140 and miR-136 under PTX exposure in vivo. (E and F) The mRNA and protein expressions of CBX2 were uncovered by qRT-PCR and western blot, respectively, in forming tumors from sh-NC, sh-NC + PTX, sh-circ_0061140 or sh-circ_0061140 + PTX group. *P < 0.05