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. 2021 Oct 16;12(11):952. doi: 10.1038/s41419-021-04245-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A Survival analysis of glioma patients with high or low expression of TOMM6, ZNRF3, EEF1G, ADORA2A, KIAA0408, KREMEN1, and XBP1. B RMRP level in the nucleus and cytoplasm of U251 and LN229 cells was measured by RT-qPCR assay after the isolation of nuclear and cytoplasmic RNA (n = 3). C, D The effects of RMRP knockdown on ZNRF3 mRNA and protein expression were measured by RT-qPCR and western blot assays at 48 h after transfection in U251 and LN229 cells (n = 3). E The effect of RMRP knockdown on ZNRF3 mRNA stability was examined through ActD and RT-qPCR assays in U251 and LN229 cells (n = 3). **P < 0.01.