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. 2021 Oct 18;13:192. doi: 10.1186/s13148-021-01125-2

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Canonical and non-canonical Wnt pathway molecules during osteoclastic differentiation, and after SETD2 overexpression and knockdown. a Western blot detection of Wnt signaling molecules during osteoclastic differentiation of RAW264.7 cells. b Detection of WNT5a and WNT3a molecules in cells during osteoclastic differentiation by immunofluorescence method. C. Western blot detection of Wnt signaling molecules after knockdown and overexpression of SETD2 in RAW264.7 cells. d Detection of WNT5A molecules in cells after overexpression and knockdown of SETD2 by immunofluorescence method. (Scale bar is 10 μM)