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. 2021 Oct 18;16:622. doi: 10.1186/s13018-021-02756-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 10 — GSEA. A Gene set related to SNI in adult group. B Gene set related to SNI in neonate group. Top 10 functional gene sets enriched in SNI status between SNI samples and NON-SNI samples are shown. C Gene set related to each high expression in each hub genes. Sankey plot showed that high expression in each hub genes were mainly enriched in pathways associated with the Lysosome pathway, the Chemokine signaling pathway, and the Neurotrophin signaling pathway