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. 2021 Oct 18;16:105. doi: 10.1186/s13020-021-00517-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Outcomes of GAP43 knockout on cerebral damage and cell survival in HI rats. a, b The gross anatomical characteristic of rat brain surface and TTC staining slices after HI in the GAP43^+/+ and GAP43^−/− groups, scale bar = 1 cm. c Comparison of brain infarction ratio among the sham, GAP43^+/+, GAP43^−/− groups. d Brain swelling comparison in the above-mentioned three groups. e TUNEL/DAPI staining images of cortex and hippocampus in the GAP43^−/− rats, scale bar = 100 μm. f Cell apoptosis rate of the left and right cortex in the GAP43^−/− rats. g Cell apoptosis rate of the left and right hippocampus in the GAP43^−/− rats. HI hypoxia ischemia, GAP43^+/+ wild-type rat, GAP43^−/− GAP43 knockout. ***p < 0.001, n = 5/group