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. 2021 Oct 12;10(1):1987636. doi: 10.1080/2162402X.2021.1987636

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© 2021 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — PRG subtypes and clinicopathological and biological characteristics of two distinct subtypes of samples divided by consistent clustering. (a) Interactions among PRGs in CRC. The line connecting the PRGs represents their interaction, with the line thickness indicating the strength of the association between PRGs. Green and pink represent negative and pink positive correlations, respectively. (b) Consensus matrix heatmap defining two clusters (k = 2) and their correlation area. (c) PCA analysis showing a remarkable difference in transcriptomes between the two subtypes. (d) Univariate analysis showing 48 PRGs related to the RFS time. (e) Differences in clinicopathologic features and expression levels of PRGs between the two distinct subtypes. PRG, pyroptosis-related gene; CRC, colorectal cancer; PCA, principal components analysis; RFS, recurrence-free-survival