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. 2021 Oct 19;11:20684. doi: 10.1038/s41598-021-00216-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Simplified schematic of findings and proposed status of Arg-involving pathways related to NO in heart, circulation, liver and kidney of O-ZSF1 and L-ZSF1 rats. Top—Enzymes (green) with substrates and metabolites (white). Bottom—Analyzed rat organs and tissues with arrows indicating the changes (p < 0.05) compared to L-ZSF1. ↑ higher in O-ZSF1, ↔ no differences, ↓ lower in O-ZSF1. Arrow in brackets indicates a trend (p < 0.1). *Orn/Cit were measured together in Urine. iNOS and Arginase were not determined in cardiac macrophages directly—the depicted regulation is hypothesis generating only.