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. 2021 Oct 19;7:299. doi: 10.1038/s41420-021-00689-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A The inhibitory effect of C-Raf/ERK pathway after treatment with Raf inhibitor LY3009120 in C33A cells. B, C CCK-8 assay and colony formation assay showed that LY3009120 reversed TM7SF2-induced promotion of proliferation in C33A cells. D The flow cytometry assay showed that LY3009120 treatment rescued the suppression of apoptosis by TM7SF2 overexpression in C33A cells. *p < 0.05 compared to control cDNA, #p < 0.05 compared to control cDNA+ LY3009120, Δp < 0.05 compared to TM7SF2 cDNA.