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. 2021 Jul 17;106(11):3213–3227. doi: 10.1210/clinem/dgab526

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© The Author(s) 2021. Published by Oxford University Press on behalf of the Endocrine Society.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (https://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 4. — Analysis of expression of HDAC3, 6, 8, 9, and 11 by PBMCs from patients with AITD and controls. A) Blood samples were obtained from healthy controls (n = 13) and patients with HT (n = 12), GD (n = 15), nonautoimmune hypothyroidism (n = 8), or hyperthyroidism (n = 10) and their PBMCs were analyzed by RT-qPCR for the levels of HDAC3, 6, 8, 9, and 11 mRNA. No significant differences were detected among the groups. B) Western blot analysis of HDAC9 expression in PBMCs homogenates from the indicated individuals. A significantly higher amount of HDAC9 was observed in samples from patients with AITD. *P < 0.05.