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. 2021 Oct 21;12:6115. doi: 10.1038/s41467-021-26347-z

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Comparison of IL33 expression between genotypes for SNPs rs10975479, rs1888909, and rs992969 from bronchial epithelial cells from 124 asthmatic and non-asthmatic adult subjects, mostly of European ancestry (upper panels) and nasal epithelial cells from 189 African-American children from high risk asthma families (lower panels). b Comparison of IL-33 cytokine levels between genotypes for SNPs rs10975479 (n = 27), rs1888909 (n = 27), and rs992969 (n = 25) measured in plasma from Hutterite children (all European ancestry). The asthma-associated risk allele at each SNP is highlighted in red (x-axis). The number of subjects per group is shown below the genotype. Boxplot center line: median; box limits: upper and lower quartiles; whiskers: 1.5× interquartile range. Statistical significance was determined using an additive linear model. Source data are provided as a Source Data file.