Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2021 Sep 27;10(10):967. doi: 10.3390/biology10100967

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

HCQ (60 μM for 48h) treatment modulated to the acute INF (LPS and 1 μg/mL for 16h)-caused upregulation of TRPV1 current densities in the U937 monocytes. (n = 3–6 and mean ± SD). The TRPV1 was activated by CAP (10 μM), although it was blocked by CPZ (100 μM). W.C.: whole cell. (A) Control (Cntr) (without CAP treatment). (B) Cntr + CAP group with the treatments of CAP and CPZ. (C) Acute INF + CAP group with the treatment of CAP and CPZ after the LPS treatment. (D). HCQ + CAP with CAP treatment after the HCQ incubation. (E) HCQ + acute INF + CAP group with the treatment of CAP and LPS after the HCQ treatment. (F) The mean densities of TRPV1 current. (G). Image of whole cell configuration. (^a p ≤ 0.05 vs. Cntr. ^b p ≤ 0.05 vs. the group of Cntr + CAP. ^c p ≤ 0.05 vs. INF+CAP).