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. 2021 Oct 15;9(10):1477. doi: 10.3390/biomedicines9101477

Table 4.

The antibiotic susceptibility patterns, pilus distribution and presence of ISSag5 in all serotype III GBS isolates.

No. (%) of All 190 Type III GBS Isolates
ISSag 5 Presence of ISSag 5 (Total n = 33, 17.4%) Absence of ISSag 5 (Total n = 157, 82.6%)
Pilus genes PI-1 + PI-2a PI-1 + PI-2b PI-2b PI-1 + PI-2a PI-1 + PI-2b PI-2b
Total n (%) 4 (2.1%) 29 (15.3%) 0 (0) 46 (24.2%) 0 (0) 111 (58.4%)
Sequence types
ST-17 (total n = 128) 0 (0) 24 (82.8) - 0 (0) - 104 (93.7)
Non-ST17 (total n = 62) 4 (100) 5 (17.2) - 46 (100) - 7 (6.3)
Sources
Neonatal invasive diseases 1 (25.0) 19 (65.5) - 12 (26.1) - 86 (77.5)
Adult invasive diseases 1 (25.0) 6 (20.7) - 29 (63.0) - 11 (9.9)
Maternal colonization 2 (50.0) 4 (13.8) - 5 (10.9) - 14 (12.6)
ICESag37
Presence 0 (0) 3 (10.3) - 8 (17.4) - 105 (94.6)
Absence 4 (100) 26 (89.7) - 38 (82.6) - 6 (5.4)
Antibiotic resistance patterns (No. (%) of resistant GBS isolates)
Ery (R) + Clin (R) 0 (0) 7 (24.1) - 15 (32.6) - 106 (95.5)
Ery (S) + Clin (S) 4 (100) 15 (51.7) - 23 (50.0) - 3 (2.7)
Ery (R) + Clin (S) 0 (0) 7 (24.1) - 3 (6.5) - 0 (0)
Ery (S) + Clin (R) 0 (0) 0 (0) - 5 (10.9) - 2 (1.8)

Ery: erythromycin; Clin: clindamycin; R: resistant; S: susceptible. All GBS isolates are susceptible to vancomycin, teicoplanin, ampicillin, penicillin, and cefotaxime; - means zero.