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. 2021 Oct 25;40:337. doi: 10.1186/s13046-021-02129-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — TMEM44-AS1 promotes glioma cell growth in vivo. (A-C) Glioma cells stably transfected with either sh-TMEM44-AS1 or sh-con were inoculated into the flanks of nude mice to construct a transplanted tumor model. Representative images of tumor volume growth curve and tumor weight were shown. (D) RT-qPCR was conducted to detect the expression levels of TMEM44-AS1 in tumor samples. **P < 0.01. (E) Ki67 and PCNA immunohistochemical (IHC) staining of tumor tissues of mice injected with sh-con or sh-TMEM44-AS1 cells. (F) The Ki67 and PCNA positive cells were qualified. **p < 0.01 vs. sh-con