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. 2021 Oct 12;11:725336. doi: 10.3389/fonc.2021.725336

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Copyright © 2021 Zhang, Ma, Long, Sun, Liu, Lin and Zhao

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Runx3 knockdown inhibits the expression levels of genes involved in DNA repair and anti-apoptosis pathways in AML cells. (A–C) The relative mRNA expression level of DNA repair (A), antiapoptosis (B), and leukemogenesis (C) related genes that RUNX3 binds to only in AML cells. Data represent mean ± SEM of six mice. **p < 0.01, ***p < 0.001. ns, not significant.