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. 2021 Oct 13;15:743546. doi: 10.3389/fnins.2021.743546

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Copyright © 2021 Niu, Tong and Blevins.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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A schematic of circuitry that potentially contributes to the effectiveness of CNS OT on energy homeostasis. OT release within the CNS and spinal cord (shown in blue arrows) or periphery (shown in gray) may impact metabolic processes that result in the reduction of body weight. Dotted arrow represents implicated pathways from NTS to spinal cord and from sympathetic preganglionic neurons in the spinal cord to BAT and WAT. ARC, arcuate nucleus; BAT, brown adipose tissue; CeA, central nucleus of the amygdala; DMV, dorsal nucleus of the vagus; NaCC, nucleus accumbens; NTS, nucleus of the solitary tract; OT, oxytocin; PBN, parabrachial nucleus; PP, posterior pituitary; PVN, paraventricular nucleus; SON, supraoptic nucleus; VTA, ventral tegmental area; WAT, white adipose tissue.