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. 2021 Oct 19;24(6):862. doi: 10.3892/mmr.2021.12502

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Copyright: © Chai et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — PLEK2 modulates apoptosis in colorectal carcinoma cells. (A) HCT-116 and (B) RKO cells transfected with siCtrl, siPLEK2#1 and siPLEK2#2 were stained with PI/Annexin V-FITC and cell apoptosis was analyzed using a flow cytometry system. Left, apoptosis graphs. Right, quantitative analysis of cell apoptosis. Ctrl and PLEK2-OE (C) HCT-116 and (D) RKO cells were stained with PI/Annexin V-FITC and cell apoptosis was analyzed using a flow cytometry system. Left, apoptosis graphs. Right, quantitative analysis of cell apoptosis. **P<0.01; ***P<0.001 vs. siCtrl or Ctrl. Ctrl, emptry vector; PLEK2-OE, pleckstrin 2 overexpression; si/siRNA, small interfering RNA; siCtrl, negative control siRNA.