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. 2021 Sep 24;59(5):90. doi: 10.3892/ijo.2021.5270

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Copyright: © He et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Molecular mechanisms of acquired 3rd-generation EGFR-TKI resistance. act, activation; NTRK1, neurotrophic receptor tyrosine kinase 1; ALK, anaplastic lymphocyte kinase; ROS1, c-ros oncogene 1, receptor tyrosine kinase; STAT3, signal transducer and activator of transcription 3; EMT, epithelial-mesenchymal transition; SCLC, small cell lung cancer; SCC, squamous cell carcinoma; MET, hepatocyte growth factor receptor; HER2, human epidermal growth factor receptor 2; FGFR, fibroblast growth factor receptor; IGF1R, insulin-like growth factors 1 receptor; AXL, anexelekto.