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. 2021 Oct 20;12:758468. doi: 10.3389/fphar.2021.758468

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Copyright © 2021 Wang, Li, Xia, Guo, Miao and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Metabolic activation of emodin induced hepatotoxicity. Emodin formed oxidated intermediates metabolites, these reactive metabolites captured with GSH, and produced GSH adduct, which led to GSH exhaustion.