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. 2021 Nov 3;7:333. doi: 10.1038/s41420-021-00725-4

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Transwell assay showed that knockdown of piR-57125 promoted 786-O and OSRC-2 cell metastasis. Data represent mean ± S.D. from three independent experiments. b Wound healing assay demonstrated that knockdown of piR-57125 enhance 786-O and OSRC-2 cells’ metastatic ability. Data represent mean ± S.D. from three independent experiments. c Transwell assay showed that the metastatic ability of ccRCC cells was inhibited when piR-57125 was overexpressed. Data represent mean ± S.D. from three independent experiments. d wound healing assay revealed that overexpression of piR-57125 slowed the metastatic rate of 786-O and OSRC-2 cells. Data represent mean ± S.D. from three independent experiments. *p < 0.05; **p < 0.01; ***p < 0.001.