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. 2021 Nov 3;7:333. doi: 10.1038/s41420-021-00725-4

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a, b Transwell and wound healing assays revealed the effect of CCL3 knockdown on 786-O and OSRC-2 cells metastasis induced by knockdown of piR-57125. Data represent mean ± S.D. from three independent experiments. c, d Transwell and wound healing assays demonstrated the effect of overexpression of CCL3 on 786-O and OSRC-2 cells metastasis induced by overexpression of piR-57125. Data represent mean ± S.D. from three independent experiments. *p < 0.05; **p < 0.01; ***p < 0.001.