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. 2021 Oct 11;53(10):1569–1579. doi: 10.1038/s12276-021-00684-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Immunostaining images for PRMT1 in VSMCs of the human artery and mouse aorta. Scale bar: 40 μm. b Boxplots showing the expression of PRMTs in aortic specimens from either healthy donors (n = 10, green) or patients with abdominal aortic aneurysm (AAA, n = 49, red) (GSE57691). c Comparison of the expression of PRMT1, Myocardin (MYCD), and contractile genes in aortas from either the young (20–30 s, n = 38, light blue) or the old (60–70 s, n = 63, blue) group of human aortas (GTEx). Statistical significance was determined with two-tailed Student’s t-tests (b) or Mann–Whitney U tests (c). *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001; n.s. not significant.