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. 2021 Oct 27;53(10):1636–1646. doi: 10.1038/s12276-021-00686-9

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a Compared with the control individuals, premenopausal breast cancer was enriched with pathways contributing to the abundance of the microbiome against the steroid-related (MetaCyc pathway) and oncogenic-related pathway (KEGG pathway). b Compared with premenopausal breast cancer patients, postmenopausal breast cancer patients exhibited greater enrichment in the steroid-related pathway (KEGG pathway).