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. 2021 Oct;9(19):1500. doi: 10.21037/atm-21-4602

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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PPARγ is the target of Sch B against liver fibrosis. (A) Immunofluorescence staining of PPARγ and DAPI in liver tissue, magnification: ×200. (B) Immunofluorescence intensity of PPARγ in liver tissue (%). (C) Three-dimensional diagram of Sch B binding with PPARγ. (D) Two-dimensional diagram of Sch B in the active site of PPARγ. ^###, P<0.001 versus the control group; ***, P<0.001 versus the model group. PPARγ, peroxisome proliferator-activated receptor gamma; Sch B, Schisandrin B; DAPI, 4',6-diamidino-2-phenylindole.