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. 2021 Apr 29;19(5):665–678. doi: 10.2174/1570159X18666200814180218

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© 2021 Bentham Science Publishers

This is an open access article licensed under the terms of the Creative Commons Attribution-Non-Commercial 4.0 International Public License (CC BY-NC 4.0) (https://creativecommons.org/licenses/by-nc/4.0/legalcode), which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

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Fig. (1) — Schemic representation of the proposed SFK-converged signalling pathways in migraine pathogenesis, including NMDA receptor, P2X₇ receptor and Na⁺,K⁺-ATPase signallings that have identified relationships with SFK in migraine models (red arrows). The SFK-transmitted signallings lead to changes in cortical susceptibility to CSD, CGRP release, neuroinflammation and trigeminovascular activation (black arrows). (A higher resolution / colour version of this figure is available in the electronic copy of the article).