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. 2021 Nov 9;19:168. doi: 10.1186/s12958-021-00851-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Schematic illustrating ICA’s protection against diabetes mellitus induced testicular dysfunction in rats possibly via enhancing cell proliferation and inhibiting intrinsic mitochondria dependent apoptotic signaling. Red arrows in T2DM panel mean deleterious effects (with significant difference) to reproductive function and relative parameters when compared with normal control group (nondiabetic). Red and green arrows in ICA treatment panel represent that there are no and obvious effectiveness respectively after ICA administration when compared with T2DM group