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. 2021 Feb 20;35(6):1281–1290. doi: 10.1007/s10557-021-07151-9

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Proliferation, adhesion, and wound migration of coronary endothelial outgrowth cells in vitro. Coronary endothelial outgrowth (CEO) cells had a higher population doubling time (PDT) during the first 9 passage in comparison to human coronary artery endothelial cells (HCAECs) and human umbilical vein endothelial cells (HUVECs) (one-way analysis of variance P = 0.017; Bonferroni post-test *P < 0.05 and **P < 0.01, respectively. n = 3–9) (a) and a lower cumulative population doubling level (CPDL) when maintained long term in culture (P < 0.0001; Bonferroni post-test ***P < 0.001 and ****P < 0.0001, respectively. n = 3–9) (b). CEO cells had a similar capacity to adhere to a collagen substrate in vitro (P = 0.662. n = 3–10) (c), but following the infliction of a linear wound in the endothelial cell monolayer, CEO cells had a reduced capacity to migrate in vitro in comparison to HUVECs (P = 0.008; Bonferroni post-test ****P < 0.0001. n = 3–7) (d)