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. 2021 Nov 10;7:347. doi: 10.1038/s41420-021-00740-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a SNORA42 expression level in HCCLM9 and SK-Hep1 cells with SNORA42 knockdown. b Flow cytometry plots showing the cell cycle distribution of the indicated groups. c Percentage of apoptotic cells in the control and SNORA42-knockdown HCCLM9 and SK-Hep1 cells. d Proliferation rates of control and SNORA42-knockdown HCCLM9 and SK-Hep1 cells. e Representative images of colonies formed by HCCLM9 and SK-Hep1 cells after SNORA42 knockdown. f EdU incorporation in the indicated groups. g, h Representative images of the Transwell cell invasion assay (g) and wound healing assay (h) showing the invasion and migration rates of HCCLM9 cells and SK-Hep1 cells after SNORA42 knockdown. *p < 0.05; **p < 0.01.