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The mechanistic rationale of SARS-CoV-2 elicited acute pancreatitis. A. Differential expression patterns of SARS-CoV-2 receptors in pancreatic cells can allow viral entry and thereby initiate acute pancreatitis or exacerbate an existing one; B. SARS-CoV-2 elicited lipotoxicity can initiate a pro-inflammatory cytokine storm to further aggravate acute pancreatitis, triggering acute respiratory distress syndrome and multiorgan failure.
