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. Author manuscript; available in PMC: 2022 Jun 28.
Published before final editing as: J Neurol Neurosurg Psychiatry. 2020 Dec 28:jnnp-2020-325121. doi: 10.1136/jnnp-2020-325121

Figure 3. Evolution of brainstem lesions in MOGAD, AQP4-IgG-NMOSD, and MS.

Figure 3.

A: A MOGAD pediatric patient had a diffuse T2 FLAIR hyperintense pontine lesion on axial images (A1, arrow) that completely resolved at 6 month follow up (A2). B: An AQP4-IgG-NMOSD seropositive adult patient had a T2 FLAIR hyperintense dorsal pontine lesion on axial images (B1, arrow) that partially resolved at 9 months follow-up (B2, arrow). C: An adult patient with MS had a hyperintense lateral pontine lesion on axial T2 FLAIR images (C1, arrow) that persisted at 11 months follow up (C2, arrow).

Key: AQP4-IgG-NMOSD, aquaporin-4-IgG positive neuromyelitis optica spectrum disorder; FLAIR, fluid-attenuated inversion recovery, MOGAD, myelin oligodendrocyte glycoprotein antibody associated disorder; MS, multiple sclerosis.