Skip to main content
. 2021 Nov 12;29:100819. doi: 10.1016/j.ymgmr.2021.100819

Table 1.

Congenital hypopituitarism with POU1F1 gene mutation.

Writer, publish date Study type and case Gene mutation of POU1F1 Sex Age at diagnosis (year) Presentation Influenced hormone MRI Follow up
Jullien et al., 2021 Cohort study Homozygous c.92dup (p.Ala32Cysfs42) GH, TSH, PRL
Li et al., 2020 Case report Heterozygous c.767-769del (p.Glu256del) M 0.8 Failure to thrive, hypoglycemia, poor appetite, constipation GH, TSH, PRL
Chen, Zhang, Wu, & Li, 2019 Case report Heterozygous c.889C > T (p.R297W) F 2.3 Failure to thrive, midline facial defects GH, TSH APH Catch-up growth
Birla et al., 2019 Cohort study C.605-1G > A
C.605delC Failure to thrive GH, TSH
C.1–59 T > A
C. + 8C > T
Blum et al., 2018 Cohort study
Sibling 1 Homozygous c.427C > T (p.Arg143Ter) F 3.0 Failure to thrive, hypoglycemia, prominent forehead, late dentition GH, TSH
Sibling 2 Homozygous c.427C > T (p.Arg143Ter) F 2.0 Failure to thrive GH, TSH
Bas et al., 2018 Case series
1 sporadic patient Homozygous c.731 T > G (p.I244S) M 1.7 Failure to thrive, precocious puberty at 7-year-9-month-old GH, TSH, PRL APH Catch-up growth
1 sporadic patient Homozygous c.10C > T (p.Q4*) M 0.5 Failure to thrive, precocious puberty at 10-year-old GH, TSH, PRL APH Catch-up growth
Takagi et al., 2017 Case report
Sibling 1 Heterozygous c.143-83A > G F 0.3 Failure to thrive GH, TSH, PRL APH Catch-up growth
Sibling 2 Heterozygous c.143-83A > G F 0.3 Failure to thrive GH, TSH Normal Catch-up growth
Bertko et al., 2017 Cohort study Homozygous c.-1387_214 + 455del M 11.5 Failure to thrive GH, TSH, PRL APH
Sobrier et al., 2016 Case series of 9 patients Heterozygous c.227C > T (p.Pro76Leu) Failure to thrive GH APH or normal
Birla et al., 2016 Case control study
3 familys, 5 patients Homozygous/heterozygous c.605-1G- > A 5 M 1.6–9 Failure to thrive GH, TSH APH
1 family, 1 patient Homozygous c.605delc M 5.0 Failure to thrive GH, TSH APH
2 familys, 2 patients Heterozygous c.1-59 T > A 2 M 15–18 Failure to thrive GH, TSH, FSH, LH Normal
1 family, 1 patient Heterozygous c. + 8C- > T M 8.0 Failure to thrive GH, TSH EPP
De Rienzo et al., 2015 Cohort study: 1 patient Homozygous IVS2-3insa F 0.5 Failure to thrive GH, TSH, PRL APH
Bas et al., 2015 Cohort study
1 family, 2 patients Homozygous p.V153F 1 F, 1 M 0.9–4.1 Failure to thrive GH, TSH, PRL APH
1 sporadic patient Homozygous p.I244S M 2.0 Failure to thrive GH, TSH, PRL, FSH, LH APH
1 sporadic patient Homozygous p.Q4X M 0.5 Failure to thrive GH, TSH APH
1 sporadic patient Homozygus Ex1-2 deletion M 0.6 Failure to thrive GH, TSH, PRL APH
Turton, Strom, Langham, Dattani, & Le Tissier, 2012 Case report Compound heterozygosity: IVS1 + 3 nt(A > G)/c.793C > T (p.R265W) M 8.0 Failure to thrive, global developmental delay, midline facial defect, micropenis GH, TSH, PRL APH Catch-up growth, improved neurodevelopment
Tenenbaum-Rakover, Sobrier, & Amselem, 2011 Case report Homozygous c.502inst (p.Thr168IlefsX7) M 0.8 Failure to thrive, prolonged jaundice, constipation, hypoglycemia, seizure, midline facial defect, micropenis, delay puberty GH, TSH, PRL APH Short stature
Lee et al., 2011 Case report Homozygous c.698 T > C (p.F233S) F 2.2 Failure to thrive, developmental delay, hypotonia, poor oral intake GH, TSH, PRL Sunken diaphragma sella Poor compliance, Kocher-Debre-Semelaigne syndrome
De Graaff et al., 2010 Cohort study: 1 patient P.R271W M GH, TSH, PRL
Snabboon et al., 2008 Case report Homozygous IVS4 + 1G > A M 18.0 Failure to thrive, midline facial defect, cretinism GH, TSH, PRL APH Poor compliance, delayed psychomotor development, severe short stature
Miyata et al., 2006 Case report Homozygous p.S179R M 4.0 Failure to thrive, poor feeding GH, TSH, PRL APH Short stature
Turton et al., 2005 Cohort study
1 family, 2 patients Compound heterozygosity: c.688G > A (p.E230K)/c.515G > A (p.R172Q) 2 M 0.1–0.3 Failure to thrive GH, TSH, PRL APH
2 familys, 2 patients Homozygous c.688G > A (p.E230K) 1 F, 1 M 1.4–2.3 Failure to thrive GH, TSH, PRL APH Catch-up growth
3 familys, 5 patients Heterozygous c.811C > T (p.R271W) 2 F, 3 M 0.2–6 Failure to thrive GH, TSH, PRL APH or normal Short stature
1 sporadic patient Compound heterozygosity: c.688G > A (p.E230K)/ins778a F 2.4 Failure to thrive GH, TSH, PRL APH
Rainbow et al., 2005 Cohort study
1 family, 2 patients Heterozygous p.R271W 2 F 0.9–1.3 Failure to thrive, optic nerve hypoplasia GH, TSH, PRL APH
1 couple of twins patients Homozygous p.F233L 2 F At birth Intrauterine growth retardation, midline facial defect, short upper limbs, poor feeding GH, TSH Expired at 3-week-old and 2-month-old
Ward et al., 1998 Case report Heterozygous c.811C > T (p.R271W) M 1.7 Failure to thrive, prolonged jaundice, constipation, hypoglycemia, developmental delay, midline facial defect GH, TSH, PRL Normal
Rodrigues Martineli, Braga, De Lacerda, Raskin, & Graf, 1998 Case report Heterozygous c.811C > T (p.R271W) F 13.0 Failure to thrive, midline facial defect, normal psychomotor development GH, TSH, PRL APH Poor compliance, short stature, hypothyroidism
Fofanova et al., 1998 Cohort study Heterozygous p.P14L F 3.8 Failure to thrive GH, TSH, PRL Catch-up growth
Arnhold et al., 1998 Case report Heterozygous p.R271W F 14.0 Failure to thrive, normal psychomotor development, delay pubarche GH, TSH, PRL Reduce size of pituitary Short stature
Pfaffle et al., 1992 Cohort study: 2 patients Homozygous c.527C > G (p.A158P) 1 F, 1 M GH, TSH, PRL Normal

M: male; F: female; GH: growth hormone; TSH: thyroid stimulating hormone; PRL: prolactin; FSH: follicle-stimulating hormone; LH: luteinizing hormone; APH: Anterior pituitary hypoplasia.