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. 2021 Nov 17;14:162. doi: 10.1186/s13048-021-00907-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Signaling pathways involved in the treatment with ghrelin and cisplatin. A-B Western blotting analysis of the expression of p-ERK1/2 and p-p38 under treatment with ghrelin in time-dependent (10 nM) (A) and dose-dependent (B) manners for 20 min. C Western blotting analysis of the expression of p-ERK1/2 and p-p38 with the treatment of cisplatin for 12 h in dose-dependent manners. D Western blotting analysis of the expression of p-CDK1, cyclin B1, and p21 in HO-8910 PM treated with MAPK inhibitors (U0126 or SB202190, 5 μM, respectively), cisplatin and the combination of inhibitors with cisplatin for 12 h. Data are shown as the mean ± SD from three independent experiments. *p < 0.05