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. 2021 Oct 29;48(12):7853–7863. doi: 10.1007/s11033-021-06807-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — SSd alleviated hepatic fibrosis in CCl₄-treated mice. Sirius red (A) and Masson staining (B) for liver tumors in control mice (n = 5), CCl₄-treated mice (n = 5) and CCl₄-SSd-treated mice (n = 5). IHC for Col1α1 (C) and α-SMA (D) levels in liver tumors of control mice (n = 5), CCl₄-treated mice (n = 5) and CCl₄-SSd-treated mice (n = 5). Scale bars = 200 μm for Sirius red, Masson and IHC. Below, five images of each liver and five livers from different mice were quantified for each group. **p < 0.01. (Color figure online)