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. 2021 Oct 29;48(12):7853–7863. doi: 10.1007/s11033-021-06807-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — SSd alleviated liver fibrosis through regulating GPER1/autophagy pathway. A LX-2 cells viability was assayed using CCK-8 after TGF-β treatment in the absence or presence of SSd, G15 or Rapa. B qPCR analysis of α-SMA and Col1α1 mRNA expression level in TGF-β-activated LX-2 cells treated with SSd in the absence or presence of G15 or Rapa. C, D Western blot analysis for α-SMA in TGF-β-activated LX-2 cells treated with SSd in the absence or presence of G15 or Rapa. *p < 0.05, **p < 0.01