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. 2021 Nov 23;12(12):1102. doi: 10.1038/s41419-021-04273-8

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A Co-IP verification of ACTN3-ACSL3 and ACTN3-LPCAT1 interactions. B ACTN3 was truncated according to the domain. The Co-IP detection of the interactions of ACSL3/LPCAT1 and different ACTN3 domains. C The schematic diagram of ACTN3 recruited on LD. D The rate of actin-filament remodeling in loaded cells with siACSL3 and siLPCAT1 and unloaded cells, bar, 20 μm. E The count of microfilament number per cell. *p < 0.05. Results are from three technical repeats (N = 3) for a representative of three biological repeats (N = 3).