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. 2021 Nov 24;8(2):e001842. doi: 10.1136/openhrt-2021-001842

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© Author(s) (or their employer(s)) 2021. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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CML and NOX2 accumulation in the cerebral vasculature show no correlation. The amounts of CML-positive and NOX2-positive blood vessels per square centimetre were tested for correlation within the non-MI control group (A) and the group of patients with MI (B), the latter being further divided into the different phases after MI infarction: 3–6 hours after MI (P1, C), 6 hours–5 days after MI (P2, D) and 5 to 14 days after MI (P3, E). A linear regression was used for analysis. CML, N^ε-(carboxymethyl)lysine; IH, immunohistochemical; MI, myocardial Infarction; NOX2, NADPH oxidase 2.