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. 2021 Nov 3;11(11):1629. doi: 10.3390/biom11111629

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Intracellular and extracellular endogenous activators (A) and inhibitors (B) of BKCa channels. BKCa: Big/large conductance calcium activated potassium channels; NO: Nitric oxide; CO: Cabon monoxide; GC: Guanylyl cyclase; cGMP: Cyclic guanosine monophosphate; PKG: Protein Kinase G; H₂O₂: Hydrogen peroxide; K⁺: Potassium ion; Fe²⁺: Ferrous ion; Mg²⁺: Magnesium ion; EET: Epoxyeicosatrienoic acid; HETE: Hydroxyeicosatrienoic acid; ER: Estrogen receptor; SR: Sarcoplasmic reticulum; Ca²⁺: Calcium ion; GPCR: G-Protein coupled receptor; CGRP: Calcitonin gene related peptide; CLR: Calcitonin receptor-like receptor; AC: Adenylyl cyclase; PPAR: Peroxisome proliferator activated recetor; cAMP: Cyclic adenosine monophosphate; PKA: Protein kinase A. Ang II: Angiotensin II; ET-1: Endothelin-I; PLC: Phospholipase C; DAG: Diacylglycerol; PKC: Protein kinase C; c-srci: Tyrosine kinase src (inactivated); GSH: Glutatione; NADH: Reduced nicotinamide adenine dinucleotide; OONO⁻: Peroxynitrate. Figure created using the illustrations by Servier Medical Art by Srvier licensed under a Creative Commons Attribution 3.0 unported License.