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. 2021 Nov 16;10(11):2818. doi: 10.3390/foods10112818

Table 2.

Antioxidant activity and advanced glycation end products (AGEs) inhibitory potential of the red rice genotypes.

Red Rice Genotypes DPPH RSA
(% Inhibition)
FRAP
(µmol Fe2+/g)
TEACABTS
(µmol TE/g)
AGEs Formation Inhibition
(% Inhibition)
Kattuyanam 94.89 ± 0.16 e 118.68 ± 0.30 d 24.48 ± 0.21 e 68.17 ± 0.43 c
Chennangi 92.11 ± 0.08 d 99.68 ± 0.18 b 7.56 ± 0.03 b 55.30 ± 0.21 b
Karungkuruvai 86.31 ± 0.21 b 112.65 ± 0.20 c 9.94 ± 0.04 c 65.90 ± 0.79 c
Sona masuri
(White rice)
5.21 ± 0.08 a 7.61 ± 0.21 a 0.67 ± 0.03 a 5.35 ± 0.19 a
Positive controls 89.16 ± 0.11 c
(Ascorbic acid)
123.27 ± 0.16 e
(Ascorbic acid)
18.71 ± 0.06 d
(Butylated hydroxytoluene)
91.21 ± 0.81 d
(Aminoguanidine)

Data are expressed as mean ± standard deviation (n = 3). DPPH RSA—2,2-diphenyl-1-picrylhydrazyl radical scavenging activity, FRAP—Ferric reducing antioxidant potential, TEAC—Trolox equivalent antioxidant capacity, ABTS—2,2′-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid, AGEs—advanced glycation end products. Different alphabets within the same column indicate significant differences across rice varieties at p ≤ 0.05.