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. 2021 Nov 27;21:297. doi: 10.1186/s12871-021-01517-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Propofol blocked A2780 and SK-OV-3 cell malignant behaviors by sequestering S100B. A The protein level of S100B in different doses of Propofol-treated A2780 and SK-OV-3 cells was checked by western blot. B The efficiency of S100B overexpression was checked by western blot. Then, in Propofol-treated A2780 and SK-OV-3 cells transfected with S100B or pcDNA, C the protein level of S100B was checked by western blot. D Cell viability was detected by MTT assay. E Colony formation ability was detected using colony formation assay. F-H Glucose consumption, lactate production and ATP level were investigated using matched kits. I-K Cell migration was assessed by transwell assay and wound healing assay, and cell invasion was assessed by transwell assay. **P < 0.01 and ***P < 0.001