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. 2021 Nov 27;21:566. doi: 10.1186/s12872-021-02379-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — A Mean ABCA1 DNA methylation levels in EAT and SAT; B ABCA1 mRNA levels in EAT and SAT samples from the CAD and NCAD groups; C ABCA1 mRNA levels in EAT and SAT samples from the subgroup of patients with CAD and concomitant carotid artery disease or peripheral artery disease compared to the NCAD group; D ABCA1 protein levels in EAT samples from the CAD and NCAD groups and representative western blot. The origin bands of WB are presented in Additional file 5. Note: The CAD+ subgroup includes patients with CAD and concomitant carotid artery disease or peripheral artery disease