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. 2021 Nov 29;12(12):1116. doi: 10.1038/s41419-021-04406-z

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Mass spectrum analysis of protein expression changes in gastric tumor tissues (n = 45). The heatmap depicts the relative protein abundance in tumor tissues (T) and paired paracarcinoma tissues (P). B Validation of SCD1 dysregulation in GC by using western blotting analysis (n = 12). C Quantitative analysis of (B) (n = 12). D Relative levels of SCD1 mRNA in gastric tumor tissues (n = 12). E Validation of SCD1 dysregulation in GC tissues using protein microarray (n = 112). F High expression of SCD1 predicts poor overall survival in GC. Patients were divided into the SCD1 high group (n = 280) and SCD1 low group (n = 595) based on the average value of SCD1 mRNA levels. G SCD1 is negatively correlated with polyunsaturated fatty acids (PUFAs) (C20:4 and C22:4) (n = 112). H SCD1 is positively correlated with monounsaturated fatty acids (MUFAs) (C16:1 and C18:1) (n = 112). I SCD1 is negatively correlated with lipid ROS production (n = 112). *p < 0.05 and ***p < 0.001.