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. 2021 Nov 22;25(1):24. doi: 10.3892/mmr.2021.12540

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Sch A downregulates the expression levels of ANP, BNP, B-MHC and miR-155 induced by CHF. (A) mRNA expression levels of ANP, BNP and B-MHC in myocardial tissue were quantified by RT-qPCR. GAPDH was used as a control. (B) The expression of miR-155 in myocardial tissue was measured via RT-qPCR. U6 was used as a control. ***P<0.001 vs. Control; ^##P<0.01, ^###P<0.001 vs. CHF. Sch A, Schisandrin A; CHF, chronic heart failure; ANP, atrial natriuretic peptide; BNP, B-type natriuretic peptide; B-MHC, B-myosin heavy chain; miR, microRNA; RT-qPCR, reverse transcription-quantitative PCR; Sch A-H, injected intraperitoneally with 40 mg/kg/day Sch A; Sch A-L, injected intraperitoneally with 20 mg/kg/day Sch A.